About Secondary Hyperparathyroidism

About Secondary Hyperparathyroidism

SHPT is an early and major complication of CKD and progresses as GFR declines1

Parathyroid hormone and calcitriol levels as GFR declines Chart

A study evaluating calcitriol and PTH values in 150 patients with various levels of chronic renal insufficiency.

  • Parathyroid hormone levels begin to rise by CKD stage 3
    (GFR <60 mL/min/1.73 m2)2
  • Decreases in active vitamin D occur even before PTH begins to rise2
  • Patients may have already lost up to 50% of their active vitamin D levels as early as CKD stage 32

Calcium and phosphorus levels may appear normal even as PTH rises3

Prevalence of low calcium and high phosphate by GFR category Bar Graph

Prevalence data based on results of patients (N=446) enrolled in a multicenter, longitudinal cohort study of patients with chronic kidney disease. This graph was adapted from K/DOQI clinical practice guidelines for chronic kidney disease: evaluation, stratification, and classification.

  • Calcium and phosphorus levels may appear normal at earlier stages of CKD3,4
  • By CKD stage 5, mineral alterations (decreased calcium and increased phosphorus) are common4

References

  1. National Kidney Foundation. K/DOQI clinical practice guidelines for bone metabolism and disease in chronic kidney disease. Am J Kidney Dis.2003;42(suppl 3):S1-S202.
  2. Llach F, Yudd M. Pathogenic, clinical, and therapeutic aspects of secondary hyperparathyroidism in chronic renal failure. Am J Kidney Dis.1998;32(suppl 2):S3-S12.
  3. Martinez I, Saracho R, Montenegro J, Llach F. The importance of dietary calcium and phosphorus in the secondary hyperparathyroidism of patients with early renal failure. Am J Kidney Dis.1997;29:496-502.
  4. National Kidney Foundation. K/DOQI clinical practice guidelines for chronic kidney disease: evaluation, classification, and stratification. Am J Kidney Dis.2002;39(suppl 1):S1-S266.